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  • 匿名
关注:1 2013-05-23 12:21

求翻译:importantly, the transiently increased [Ca2+]i induced by 10 µM βE2 treatment for 0.5 hrs was mediated by the phosphatidylinositol-3-kinase (PI3K) and gated by the L-type voltage-gated Ca2+ channels (L-VGCC), but the increased [Ca2+]iinduced by 100 µM H2O2 treatment for 2 hrs was not affected是什么意思?

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importantly, the transiently increased [Ca2+]i induced by 10 µM βE2 treatment for 0.5 hrs was mediated by the phosphatidylinositol-3-kinase (PI3K) and gated by the L-type voltage-gated Ca2+ channels (L-VGCC), but the increased [Ca2+]iinduced by 100 µM H2O2 treatment for 2 hrs was not affected
问题补充:

  • 匿名
2013-05-23 12:21:38
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  • 匿名
2013-05-23 12:23:18
重要的是 , 快速增加 [ Ca 2 + ] i 引起的 10 μ m βE 2 治疗 0.5 小时 , 调解 phosphatidylinositol 3 激酶 ( PI EVA3K ) 和门控的 L 型电压门控 Ca 2 + 通道 ( L VGCC ) , 但增加的 [ Ca 2 + ] iinduced 100 μ m 的 H 2 O 2 处理 2 小时不受影响
  • 匿名
2013-05-23 12:24:58
重要地,我由10 (µM β) E2治疗导致0.5小时的瞬变地增加的Ca2+由磷脂酰肌醇3激酶PI3K (斡旋) 并且由L类型电压装门的Ca2+渠道L-VGCC (装门),但增加的 (Ca2+)由100 µM水2治疗iinduced 2个小时不受影响
  • 匿名
2013-05-23 12:26:38
重要的是,一剎那之间增加 [Ca2 +] 我诱导 10 µ M βE2 治疗 0.5 小时是介导磷脂酰肌醇 3 激酶 (PI3K) 和门控的 L-型电压门控 Ca2 + 通道 (L-VGCC),但增加 [Ca2 +] iinduced 由 100 µ M H2O2 治疗 2 小时不受影响
  • 匿名
2013-05-23 12:28:18
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